This preview shows page 1 out of 2 pages.

Save
View full document
View full document
Premium Document
Do you want full access? Go Premium and unlock all 2 pages.
Access to all documents
Download any document
Ad free experience
Premium Document
Do you want full access? Go Premium and unlock all 2 pages.
Access to all documents
Download any document
Ad free experience

Unformatted text preview:

Module 2:Reading assignment: Lehn Chapter 12-19ATI: NeurologyMedications to know:Atropine (IV), Oxybutynin (Dirtopan), Scopolomine, Vecuronium (Norcuron), Succinylchole (Anective), Epinephrine, Phenylephrine , Dopamine, Albuterol, epipen (p. 158). Terazosin, (Hytrin) Doxazosin (Cardura), Phentolomine (Regitine), Metopropolol, Esmolol, Carvidilol, clonidine, reserpineMost of these drugs are going to be covered in other sections. But they are meds that are used frequently so I listed them so you can note how they work as you read.Study Questions Module 21) Describe the difference between the sympathetic and parasympathetic system.2) Cholinergic receptors mediate responses to___________________3) Adrenergic receptors mediate responses to ___________________4) Location of Alpha receptors___________________________________5) Location of Alpha 2 receptors______________________________________6) Location of Beta1 receptors_______________________________________7) Location of Beta2 receptors______________________________________8) Cholinergic drugs have what side effects: 9) Muscarinic agonist and antagonist stimulate or block what system. 10) What is Bethanechol used for? What are benefits? What are risks?11) What is Atropine used for? What are the benefits? What are the risks? 12) What is Myasthania Gravis? (look it up elsewhere)13) How does a cholinesterase inhibitor work in Myasthenia Gravis? 14) What is a neuromuscular blocking agent? How does it work? What is it used for? 15) What special precautions are needed when administering a neuromuscular blocking agent? 16) What are adrenergic agonists? 17) What are the 5 receptor types. When receptors are activated what is the response. For example: Activation of alpha1 receptors causes vasoconstriction and mydriasis (dilation of the pupil). Negative response HTN, necrosis, bradycardia.18) Adrenergic antagonist block the adrenergic receptors. 19) What happens when the five receptors are blocked?20) Therapeutic reasons/cases to use an Alpha blocker21) Therapeutic reasons/cases to use a beta


View Full Document

UNCW NSG 325 - Module 2 NSG 325

Download Module 2 NSG 325
Our administrator received your request to download this document. We will send you the file to your email shortly.
Loading Unlocking...
Login

Join to view Module 2 NSG 325 and access 3M+ class-specific study document.

or
We will never post anything without your permission.
Don't have an account?
Sign Up

Join to view Module 2 NSG 325 2 2 and access 3M+ class-specific study document.

or

By creating an account you agree to our Privacy Policy and Terms Of Use

Already a member?