Study Guide: Biopysch
34 Cards in this Set
Front | Back |
---|---|
Declarative Memory
|
Explicit or episodic Facts and events
|
Nondeclarative Memory
|
Implicit Skills, habits, classical conditioning
|
Encoding
|
Classifying/categorizing experiences
|
Consolidation
|
Short term memory to long term memory
|
Retrograde Amnesia
|
Retrograde amnesia is a form of amnesia where someone will be unable to recall events that occurred before the development of amnesia.
|
Anterograde Amnesia
|
Anterograde amnesia is a loss of the ability to create memories after the event that caused the amnesia occurs.
|
MAO inhibitors
|
Block destruction of excess monoamines in terminals.
|
Tricyclic Antidepressants
|
Block reuptake of monoamines at the synapse.
|
Monoamine Hypothesis
|
Depresion involves reduced activity at norepinephrine and serotonin synapses.
|
Lithium Carbonate
|
Stabilizes neurotransmitter and receptor systems to prevent the large swings seen n manic-depressive cycling. Affects several neurotransmitter systems.
|
Diathesis
|
A predisposition to become depressed.
|
Diathesis Stress Model
|
Some indication that early exposure to stress increases the likelihood of developing depression in adulthood.
|
Serotonin Transporter
|
Monoamine transporter protein that is responsible for reuptake of serotonin into the presynaptic neuron.
|
Hyperfrontality
|
Excessive activity in prefrontal cortex and amygdala
|
Positive Symptoms of Schizophrenia
|
Delusions (false beliefs, illogical thinking), hallucinations (false sensory perceptions), disorganized speech (incomprehensible language)
|
Negative Symptoms of Schizophrenia
|
Social withdrawal, anhedonia, blunted affect (flattening of emotional expression), avolition (inability to initiate and sustain goal directed behavior), alogia (poverty of speech)
|
Phenothiazines (i.e. chlorpromazine)
|
1st class of drugs used for treatment of schizophrenia. Tranquilizers that calmed surgical patients, did not treat schizophrenia.
|
Dopamine Hypothesis
|
Schizophrenia involves excessive dopamine activity in the brain.
|
Tardive Dyskinesia
|
Tremors and involuntary movements caused by blocking of dopamine receptors in the basal ganglia. Side effect of antipsychotic drugs used to treat schizophrenia.
|
Atypical Antipsychotics (i.e. clozapine)
|
Target D2 receptors much less, so they produce motor problems only at much higher doses. Fewer side effects. Treats some negative symptoms.
|
Hypofrontality
|
Reduced activity in the frontal lobes.
|
Glutamate Theory
|
Reduced glutamate activity in the prefrontal cortex is involved in schizophrenia. Atypical antipsychotics increase glutamate levels.
|
Sympathetic Adrenal Medullary Pathway
|
1. Activation of sympathetic nervous system
2. Stimulation of multiple organs by neurotransmitters (norepinephrine and epinephrine)
3. Stimulation of adrenal medulla (ACh)
4. Release epinephrine and norepinephrine into blood stream
|
Antibodies
|
Produced by B cells of the immune system to recognize foreign antigens (invaders) and mark them for destruction.
|
T-Cells
|
White blood cells that kill invaders.
|
B-Cells
|
Fights intruders by producing antibodies that attack a particular cell type.
|
Natural Killer Cells
|
Attack and destroy certain kinds of cancer cells and cells infected with virues. Less specific in their targets than T or B cells.
|
Innate Immunity
|
No memory, anatomical barriers, phagocytes (macrophage), natural killer cells, inflammatory response (histamine release)
|
Acquired Immunity
|
Protects against specific microorganisms, memory, acquired through having disease or immunization, T cells, B cells (antibodies)
|
Dorsomedial Thalamus
|
Structure of the brain that plays a role in memory.
|
Medial Temporal Lobe System
|
H.M. Symptoms: anterograde amnesia, mild retrograde amnesia, consolidatin disrupted, hippocampus & amygdala involved
|
N.A.
|
Deficits: anterograde amnesia, encoding functions disrupted, dorsomedial thalamus involved.
|
Korsakoff's Syndrome
|
Deficits: anterograde amnesia & remote memory loss, encoding & retrieval functions disrupted, brain areas involved are widespread, including frontal cortex and mammillary bodies of diencephalon.
|
H.M.
|
Bilateral temporal lobe damage from surgery for epilepsy at 27 years old. Has difficulty learning and retaining new information (consolidation)
|