PSYC 1100 1nd Edition Lecture 11 Outline of Last Lecture I Damage to the Non Primary Association Cortex Pre Frontal Lesions Apraxia Agnosia Neglect Aphasia o Expressive Aphasia o Receptive Aphasia II Split Brain Studies III Note Visual Pathways Outline of Current Lecture I Neurons continued a Glia b White Matter c Grey Matter II Neurotransmitters III Reciprocal Inhibition IV Learning These notes represent a detailed interpretation of the professor s lecture GradeBuddy is best used as a supplement to your own notes not as a substitute Current Lecture I Neurons continued a Glia guide migrating neurons to their destinations influence neural connections shut down process of neural growth increase communication speed b White Matter myelinated axons traversing long distances either within the brain or to and from the body c Grey Matter consists of cell bodies dendrites and unmyelinated axons and the interneurons that constitute the nervous system s micro circuitry II Neurotransmitters AcH makes muscle fibers contract Serotonin sleep mood arousal GABA inhibitory transmitter Epinephrine Dopamine involved with mental disorders III Reciprocal Inhibition nerves causing one muscle to contract could simultaneously send an inhibitory message to the partnering muscle causing it to uncontract the excitement of one thing causes the inhibition of another EX in the upper arm when the flexor contracts the extensor is inhibited IV Learning a Habituation decline in an organism s response to a stimulus once the stimulus has become familiar simplest form of learning organism recognizes event as familiar but does not learn anything about the event b Dishabituation increase in responsiveness caused by the presentation of something novel c Unconditioned Response UR product of biology and triggered by a certain stimulus d Unconditioned Stimulus US stimulus independent of any learning e Conditioned Stimulus CS initially a neutral stimulus f Conditioned Response CR product of learning and triggered by a certain stimulus g Extinction the undoing of classical conditioning
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